Search results for " Adrenal hyperplasia"

showing 10 items of 12 documents

Non-classic congenital adrenal hyperplasia due to 21-hydroxylase deficiency revisited: an update with a special focus on adolescent and adult women.

2016

Background Non-classic congenital hyperplasia (NCAH) due to 21-hydroxylase deficiency is a common autosomal recessive disorder characterized by androgen excess. Objective and rationale We conducted a systematic review and critical assessment of the available evidence pertaining to the epidemiology, pathophysiology, diagnosis and management of NCAH. A meta-analysis of epidemiological data was also performed. Search methods Peer-reviewed studies evaluating NCAH published up to October 2016 were reviewed. Multiple databases were searched including MEDLINE, EMBASE, Cochrane, ERIC, EBSCO, dissertation abstracts, and current contents. Outcomes The worldwide prevalence of NCAH amongst women presen…

0301 basic medicineAdultmedicine.medical_specialtyPediatricsHirsutismAdolescentGenetic counseling030209 endocrinology & metabolismDiseaseMiscarriage03 medical and health sciences0302 clinical medicinemedicineHumansCongenital adrenal hyperplasiahirsutismMenstruation DisturbancesGynecologyPregnancyAdrenal Hyperplasia Congenitalbusiness.industry17-alpha-HydroxyprogesteroneHyperandrogenismObstetrics and GynecologyAndrogen Antagonistsmedicine.diseasePolycystic ovary030104 developmental biologyReproductive MedicineFemalebusinessInfertility FemaleHuman reproduction update
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Clinical and hormonal characteristics in heterozygote carriers of congenital adrenal hyperplasia

2020

Abstract Non-classical congenital adrenal hyperplasia (NC-CAH) includes a group of genetic disorders due to a broad class of CYP21A2 variants identifying a disease-causing ‘C’ genotype. The heterozygous carriers of CYP21 mutations are at increased risk of developing clinically evident hyperandrogenism, even though clinical and laboratory characteristics are still underestimated. With the aim of obtaining a more accurate delineation of the phenotype of heterozygous carrier of CAH, we analyzed clinical, biochemical and molecular characteristics in a cohort of Sicilian subjects. Fifty-seven females with biallelic and monoallelic CYP21A2 variants classifying NC-CAH (24) and heterozygous carrier…

0301 basic medicineHirsutismHydrocortisoneendocrine system diseasesEndocrinology Diabetes and MetabolismClinical BiochemistryPhysiologyOverweighturologic and male genital diseasesBiochemistrySettore MED/13 - Endocrinologia0302 clinical medicineEndocrinologySettore BIO/10 - BiochimicaGenotypeMedicineChildhirsutismPolycystic ovaryfemale genital diseases and pregnancy complications030220 oncology & carcinogenesisCohortMolecular MedicineFemalemedicine.symptomAdultHeterozygotecongenital hereditary and neonatal diseases and abnormalitiesAdolescentYoung Adult03 medical and health sciencesHumansCongenital adrenal hyperplasiaMolecular BiologyHeterozygous carrierAdrenal Hyperplasia Congenitalbusiness.industryHyperandrogenismCongenital adrenal hyperplasianutritional and metabolic diseasesHeterozygote advantageCell BiologyOverweightmedicine.diseaseOligomenorrhea17OHProgesterone deficiency030104 developmental biologyMutationSteroid 21-HydroxylaseHyperandrogenismbusinessThe Journal of Steroid Biochemistry and Molecular Biology
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21-Hydroxylase and 11beta-hydroxylase mutations in Romanian patients with classic congenital adrenal hyperplasia.

2005

Congenital adrenal hyperplasia (CAH) comprises autosomal recessive disorders mainly due to defects in the 21-hydroxylase (CYP21) gene.The study aimed to perform molecular characterization in 43 Romanian patients with classical CAH forms diagnosed at the Center for Genetic Diseases of the Pediatric Clinic/University Cluj (38 with 21-hydroxylase deficiency, five with 11beta-hydroxylase deficiency), to determine the frequency of mutations in the CYP21A2 gene and attempt a genotype-phenotype correlation in patients with 21-hydroxylase deficiency.Molecular analysis was performed by direct sequencing of PCR amplified products of the CYP21A2 and CYP11B1 genes.The most frequent mutation in Romanian…

AdultMalemedicine.medical_specialtyAdolescentGenotypeEndocrinology Diabetes and MetabolismClinical BiochemistryContext (language use)Biologymedicine.disease_causeBiochemistryEndocrinologyGene FrequencyInternal medicineGenotypemedicineHumansCongenital adrenal hyperplasiaSteroid 11-beta-hydroxylaseChildAllelesMutationAdrenal Hyperplasia CongenitalRomaniaBiochemistry (medical)Genetic disorder21-HydroxylaseInfantmedicine.diseaseEndocrinologyChild PreschoolMutationbiology.proteinMutation testingSteroid 11-beta-HydroxylaseFemaleSteroid 21-HydroxylaseThe Journal of clinical endocrinology and metabolism
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Ovarian suppression reduces clinical and endocrine expression of late-onset congenital adrenal hyperplasia due to 21-hydroxylase deficiency.

1994

Objective To determine the effectiveness of GnRH-agonist (GnRH-a) treatment in women with late onset congenital adrenal hyperplasia. Design Prospective assessment of GnRH-a treatment in six women with documented late-on-set congenital adrenal hyperplasia who were not preselected. Comparisons were made to previous responses in the same patients receiving dexamethasone. Eight age- and weight-matched ovulatory women served as controls. Setting Academic medical center. Intervention Baseline blood determinations before and after IV ACTH, before and after 6months of GnRH-a treatment. Estrogen and progestin replacement was begun in all women after the 3rd month of treatment. Main Outcome Measures …

Adultendocrine systemmedicine.medical_specialtyHirsutismAdolescentmedicine.drug_classOvaryDexamethasoneInternal medicineEndocrine GlandsmedicineHydroxyprogesteronesHumansCongenital adrenal hyperplasiaProspective StudiesAge of OnsethirsutismDexamethasoneTriptorelin PamoatebiologyAdrenal Hyperplasia Congenitalbusiness.industry17-alpha-HydroxyprogesteroneOvary21-HydroxylaseObstetrics and Gynecologymedicine.diseaseAndrogenmedicine.anatomical_structureEndocrinologyReproductive MedicineEstrogenbiology.proteinAndrogensFemalebusinessProgestinhormones hormone substitutes and hormone antagonistsGonadotropinsmedicine.drugFertility and sterility
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Refractory Acne and 21-Hydroxylase Deficiency in a Selected Group of Female Patients.

2009

<i>Background:</i> Excessive androgen production, suspected in women when acne is accompanied by hirsutism and menstrual irregularities, may be due to congenital adrenal hyperplasia. This inherited disorder of cortisol biosynthesis is caused in more than 90–95% of all cases by 21-hydroxylase deficiency (21-OHD). The steroid 21-hydroxylase gene <i>(CYP21)</i> has a high degree of variability. <i>Objective:</i> This study was conducted to evaluate <i>CYP21 </i>gene mutations in a selected group of women with papulopustular and comedonal acne refractory to treatment, irregular menses and hirsutism. <i>Methods:</i> 30 out of 61 women e…

Adultmedicine.medical_specialtyAdolescentDrug ResistancePhysiologyDermatologyAdrenocorticotropic hormoneYoung AdultSettore MED/38 - Pediatria Generale E SpecialisticaPapulopustularInternal medicineAcne VulgarismedicineHumansPoint MutationCongenital adrenal hyperplasiaGenetic TestingRefractory acne Excessive androgen production Non-classical 21-hydroxylase deficiency CYP21 gene mutations.AcnehirsutismAdrenal Hyperplasia Congenitalmedicine.diagnostic_testbiologybusiness.industry17-alpha-HydroxyprogesteroneACTH stimulation test21-Hydroxylasemedicine.diseasePolycystic ovaryEndocrinologybiology.proteinFemaleSteroid 21-HydroxylaseHyperandrogenismbusinessPolycystic Ovary Syndrome
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The ratio of androstenedione: 11β-hydroxyandrostenedione is an important marker of adrenal androgen excess in women

1992

To determine if the ratio of serum androstenedione (A):11 beta-hydroxyandrostenedione (OHA) would be helpful in differentiating adrenal from ovarian hyperandrogenism.Prospective study of outpatients being evaluated for hyperandrogenism.Normal women (n = 27), those with hyperandrogenic chronic anovulation (n = 25), and 7 with adult onset of congenital adrenal hyperplasia (CAH) because of 21-hydroxylase deficiency.Fasting serum between 8:00 A.M. and 9:00 A.M. Patients with hyperandrogenic chronic anovulation and CAH received dexamethasone (DEX) 2 mg for 7 days.Serum testosterone (T), unbound T, dehydroepiandrosterone sulfate (DHEAS), A, and 11 beta-OHA by radioimmunoassay.Serum 11 beta-OHA an…

Adultmedicine.medical_specialtyAdolescentmedicine.drug_classAdrenal Gland DiseasesRadioimmunoassayDexamethasoneAnovulationchemistry.chemical_compoundDehydroepiandrosterone sulfateInternal medicineAdrenal GlandsmedicineHumansCongenital adrenal hyperplasiaProspective StudiesAndrostenedioneDexamethasoneHyperplasiaAdrenal Hyperplasia Congenitalbusiness.industryHyperandrogenismAndrostenedioneObstetrics and GynecologyRadioimmunoassaymedicine.diseaseAndrogenEndocrinologyReproductive MedicinechemistryAndrogensFemalebusinessBiomarkersmedicine.drugFertility and Sterility
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Female pelvic congenital malformations. Part I: embryology, anatomy and surgical treatment.

2011

This review covers the most important female congenital pelvic malformations. The first part focuses on the embryological development of the urogenital and anorectal apparatus, morphological features, and the diagnostic and surgical approach to abnormalities. Comprehension of the embryological development of the urogenital and anorectal apparatus is essential to understand the morphology of congenital pelvic abnormalities and their surgical treatment. Congenital pelvic malformations are characterized by specific common features; the severity of which often subverts the pelvic morphology completely and makes it difficult to comprehend before surgery. The development of imaging, mainly magnet…

Adultmedicine.medical_specialtyReconstructive surgeryanorectal malformations46 XX Disorders of Sex DevelopmentAdolescentEmbryonic DevelopmentUrogenital SystemKidneyCongenital AbnormalitiesPelvisAnus Imperforatemedicinemayer-rokitansky-küster-hauser syndromeHumanscongenital adrenal hyperplasiaCongenital adrenal hyperplasiaMayer-Rokitansky-Kuster-Hauser SyndromeAbnormalities MultipleIntestine LargeMullerian Ductsmayer-rokitansky-kuster-hauser syndrome; mayer–rokitansky–kuster–hauser syndrome; mayer-rokitansky-küster-hauser syndrome; congenital adrenal hyperplasia; anorectal malformations; bladder exstrophymedicine.diagnostic_testAdrenal Hyperplasia Congenitalbusiness.industryGenitourinary systemmayer–rokitansky–kuster–hauser syndromeUterusObstetrics and GynecologyInfantMagnetic resonance imagingAnatomyAnusmedicine.diseaseSpineSurgeryBladder exstrophymedicine.anatomical_structureReproductive MedicineSomitesEmbryologyVaginamayer-rokitansky-kuster-hauser syndromeFemalebusinessbladder exstrophy
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Polycystic ovary syndrome

2016

Polycystic ovary syndrome (PCOS) affects 5-20% of women of reproductive age worldwide. The condition is characterized by hyperandrogenism, ovulatory dysfunction and polycystic ovarian morphology (PCOM) - with excessive androgen production by the ovaries being a key feature of PCOS. Metabolic dysfunction characterized by insulin resistance and compensatory hyperinsulinaemia is evident in the vast majority of affected individuals. PCOS increases the risk for type 2 diabetes mellitus, gestational diabetes and other pregnancy-related complications, venous thromboembolism, cerebrovascular and cardiovascular events and endometrial cancer. PCOS is a diagnosis of exclusion, based primarily on the p…

OvulationHirsutismmedicine.medical_specialtyendocrine system diseases030209 endocrinology & metabolismOvaryPathogenesis03 medical and health sciences0302 clinical medicineInsulin resistanceSDG 3 - Good Health and Well-beingRisk FactorsInternal medicineAcne VulgarismedicineHumansCongenital adrenal hyperplasiaObesityAbdominal obesityhirsutism030219 obstetrics & reproductive medicinebusiness.industryOvaryHyperandrogenismAlopeciaGeneral Medicinemedicine.diseasePolycystic ovaryfemale genital diseases and pregnancy complicationsEndocrinologymedicine.anatomical_structureAndrogensQuality of LifeFemalemedicine.symptomHyperandrogenismbusinessPolycystic Ovary SyndromeNature Reviews Disease Primers
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Reproductive Dysfunction in Classical and Nonclassical Congenital Adrenal Hyperplasia Due to 21-Hydroxylase Deficiency

2021

Patients with congenital adrenal hyperplasia (CAH) both with severe (classical CAH) and mild (nonclassical NCAH) forms exhibit a wide spectrum of reproductive dysfunction. In this review, only CAH cases with 21-hydroxylase deficiency (21-OHd) will be discussed, as they represent almost all of the patients in reproductive clinical settings.

congenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyendocrine system diseasesbiologybusiness.industry21-Hydroxylasenutritional and metabolic diseasesClinical settingsurologic and male genital diseasesmedicine.diseasefemale genital diseases and pregnancy complicationsCongenital adrenal hyperplasia due to 21-hydroxylase deficiencyEndocrinologyInternal medicinemedicinebiology.proteinCongenital adrenal hyperplasiabusiness
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HLADR5 and C4BQ0 high frequency and antinuclear antibodies positivity in patients with 21 hydroxylase deficiency from Campania region

1992

HLA haplotypes, complement C4 factor and factor B immonochemical concentrations and autoantibodies titer have been studied in six patients with mild congenital adrenal hyperplasia (MC-AH), in two patients with classical congenital adrenal hyperplasia (CCAH) and in their parents. A high frequency of DR5 and C4BQO alleles have been found in MCAH patients. Moreover, C4BQO allele is carried out in three out of four cases associated with DR5. In the two CCAH patients we found a B51 and a B14 allele, the last one usually described in the non classical form of the disease in population of different ethnic origin. Signs of autoimmunity in some patients and parents have been found. C4 null alleles w…

education.field_of_studyAnti-nuclear antibodybiologybusiness.industryEndocrinology Diabetes and MetabolismPopulationAutoantibody21-HydroxylaseHuman leukocyte antigenmedicine.diseaseNull alleleEndocrinologyImmunologybiology.proteinmedicineCongenital adrenal hyperplasiaAlleleeducationbusinessJournal of Endocrinological Investigation
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